Peptides for Longevity Research | Telomere, Mitochondria & Senolysis | QSC
QSC RESEARCH COMPOUND GUIDE
hTERT ActivationTelomere LengthEpitalonP21
Longevity research targets the nine hallmarks of ageing: telomere attrition, epigenetic changes, mitochondrial dysfunction, cellular senescence, loss of proteostasis, deregulated nutrient sensing, stem cell exhaustion, altered intercellular communication, and genomic instability. QSC supplies research compounds addressing multiple hallmarks — from telomerase activation to senolysis to mitochondrial biogenesis.
What is the foundational longevity peptide combination?
Khavinson et al. identified thymalin + epitalon as producing the greatest lifespan extension (24%) of any tested peptide combination in aged mice. This combination addresses immune senescence (thymalin) and telomere attrition (epitalon) simultaneously.
How does NAD+ relate to longevity research?
NAD+ declines ~50% from young to aged tissues. NAD+ is required for SIRT1/SIRT3 (deacetylases regulating mitochondrial biogenesis and DNA repair), PARP1 (DNA damage repair), and mitochondrial electron transport. NAD+ restoration reverses multiple ageing phenotypes in rodent models.
What is the difference between senolytics and senomorphics?
Senolytics (FOXO4-DRI, PNC-27) kill senescent cells selectively. Senomorphics reduce the senescence-associated secretory phenotype (SASP) — the pro-inflammatory secretome of senescent cells — without clearing the cells themselves. QSC catalog currently focuses on senolytics.
