Midas Longevity Protocol — Research Guide



12-Compound Longevity Research Protocol — 10 Hallmarks of Aging

Midas Longevity Protocol — Research Guide

The Midas Longevity Protocol is QSC’s comprehensive anti-aging research stack — 12 compounds, 120 vials, addressing the hallmarks of biological aging as defined by Lopez-Otin et al. (2013, 2023). Each compound addresses at least one recognised aging hallmark.

Compound Manifest (12 Compounds)

# Compound Class Target Research Role
1 NAD+ Mitochondrial coenzyme SIRT1-3, PARP1-2 Hallmarks: mitochondrial dysfunction, genomic instability
2 Epitalon AEDG telomerase activator hTERT Hallmark: telomere attrition — direct hTERT activation
3 GHK-Cu Copper tripeptide Fibroblast ECM/MMP Hallmarks: altered intercellular communication, ECM degradation
4 SS-31 Cardiolipin-targeting tetrapeptide Inner mitochondrial membrane Hallmark: mitochondrial dysfunction — cristae stabilisation
5 FOXO4-DRI D-retro-inverso FOXO4 peptide FOXO4-p53 senolytic Hallmark: cellular senescence — selective senescent cell apoptosis
6 Thymosin Alpha-1 Thymic immunomodulator T-cell maturation, NK cells Hallmark: disabled macroautophagy, altered intercellular communication
7 BPC-157 Gastric pentadecapeptide NO synthase, VEGF Hallmark: stem cell exhaustion, tissue repair
8 TB-500 Thymosin Beta-4 fragment G-actin Hallmark: stem cell exhaustion, inflammation
9 CJC-1295 GHRH analogue GHRH receptor Hallmark: deregulated nutrient sensing via GH/IGF-1 axis
10 Ipamorelin GHSR-1a agonist GHSR-1a GH axis — pulsatile GH restoration
11 Glutathione Master antioxidant ROS/GSH system Hallmark: oxidative stress, mitochondrial dysfunction
12 Humanin Mitochondria-encoded 21aa CNTFR/GP130/WSX-1 Hallmark: mitochondrial dysfunction, disabled autophagy

Compound Stacking Logic

The Midas Protocol maps compounds to the Lopez-Otin hallmarks of aging: Telomere attrition → Epitalon (hTERT). Genomic instability → NAD+ (PARP repair). Mitochondrial dysfunction → SS-31 (cristae) + NAD+ (sirtuins) + Humanin (CNTFR). Cellular senescence → FOXO4-DRI (senolytic). Stem cell exhaustion → BPC-157 + TB-500. Altered intercellular communication → GHK-Cu + Thymosin Alpha-1. Disabled macroautophagy → Thymosin Alpha-1 + NAD+. Deregulated nutrient sensing → CJC-1295 + Ipamorelin (GH axis restoration). Oxidative stress → Glutathione (ROS scavenging).

Frequently Asked Questions

What is the Midas Longevity Protocol?
The Midas Longevity Protocol is QSC’s 12-compound, 120-vial anti-aging research stack. Each compound is selected to address one or more recognised hallmarks of biological aging (Lopez-Otin 2013/2023). Includes NAD+, Epitalon, GHK-Cu, SS-31, FOXO4-DRI, Thymosin Alpha-1, BPC-157, TB-500, CJC-1295, Ipamorelin, Glutathione, and Humanin.
Which aging hallmarks does the Midas Protocol address?
The protocol targets: telomere attrition (Epitalon/hTERT), genomic instability (NAD+/PARP), mitochondrial dysfunction (SS-31, NAD+, Humanin), cellular senescence (FOXO4-DRI), stem cell exhaustion (BPC-157, TB-500), altered intercellular communication (GHK-Cu, Thymosin Alpha-1), deregulated nutrient sensing (CJC-1295/Ipamorelin), and oxidative stress (Glutathione).
Is the Midas Protocol suitable for women’s research?
Yes — the Midas Protocol contains no androgen-class compounds. It is gender-neutral by design. For women’s research models, the Venus Protocol offers similar longevity coverage (Epitalon, NAD+, GHK-Cu, Glutathione) with additional GLP-1 and sexual function compounds.
Research Use Only: All QSC compounds are sold strictly for laboratory research purposes only. Not for human consumption, veterinary use, or any other application. Researchers are responsible for compliance with local regulations.
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